Duchenne muscular dystrophy(DMD) is a genetic disease. Due to the gradual deterioration of muscles and the difference between muscle dystrophin protein and muscle dystrophin protein, Duchenne muscular dystrophy is necessary to maintain muscle tissue integrity. In 1860, Guillaume Benjamin Amand Duchenne, a French neuroscientist, first described Du Xinggen’s nutritional disorder. Touxing muscular dystrophy is one of the few diseases called muscular dystrophy, including Becker muscular dystrophy. The onset of DMD symptoms usually occurs when they are young. This situation mainly affects men, but girls are rarely affected. The incidence of Duchenne muscular dystrophy is close to 6 cases per 100000 people.

The main symptom of Duchenne muscular dystrophy is muscle weakness. It may also have started at the age of 2 or 3. This force actually starts to affect the proximal muscle group of the body close to the core. More distant arm or leg muscles will be affected later. Generally speaking, the lower limb muscle group is affected by the upper limb muscle group transfer. Affected children often have difficulty jumping, running and walking. Other signs and symptoms include enlarged calves, hobbling, and an inward contour of the spine. Then, the heart and respiratory muscles were affected, and there was a problem. Progressive weakness and spinal muscle weakness lead to impaired lung activity, which may eventually lead to severe respiratory failure, which may be very important. Becker muscular dystrophy is very similar to Duchenne muscular dystrophy, but its onset is generally slower and unpredictable than Duchenne muscular dystrophy in adolescence.

In 1986, researchers observed a special gene on the X chromosome with a defect(mutation) that would lead to Duchenne muscular dystrophy. The actual protein related to this gene was quickly identified and named as dystrophin. This is because muscle cells lack dystrophic proteins, making them weak and vulnerable. DMD has an X-linked genetic model of tolerance inherited from the mother of the carrier. Women as carriers have typical dystrophin genes on a single X chromosome, and abnormal dystrophin genes on the other X chromosome. Almost all DMD carriers have no such symptoms themselves.

although there is no cure for Duchenne muscular dystrophy, this treatment can extend the normal activity time of people with this disease and help strengthen the lung and heart muscles. Treatment options include medication, physical rehabilitation, occupational therapy, surgery, and other surgical procedures. The treatment group continued to evaluate the strength of walking, swallowing, breathing and hands, so as to adjust the treatment plan according to the progress of the disease. Recently, young people with DMD usually cannot live beyond the age of 10. The latest advances in heart and respiratory therapy have led to an increase in life expectancy. Many young people with DMD can go to college, get married and have children. Living to be in your 30s is now common.